Date of Award
Master of Science
Health and Sport Science
Exercise, Sport & Movement Sci
Rigidity is a symptom of Parkinson’s disease (PD) that is caused by neural and intrinsic mechanisms. Individuals with PD show increased rates of energy expenditure during walking and it is speculated that intrinsic rigidity is contributing. Purpose: Evaluate the effects of intrinsic rigidity on the metabolic cost of walking. Methods: 10 experimental subjects were used to create muscle-driven simulations of walking. During the simulations, intrinsic rigidity was increased in the model’s lower extremity musculature. Whole-body metabolic power was calculated in each state of increased intrinsic rigidity. Results: There were no significant differences (p=.448) in simulated metabolic power between states of intrinsic rigidity. Conclusion: It is speculated that the simulated musculotendon system adapted to a more optimal fiber length after alterations to intrinsic rigidity resulting in more efficient movement and that the simulated alterations were not large enough to change systemic energy expenditure at this particular walking speed.
dissertation or thesis originally submitted to the local University of Memphis Electronic Theses & dissertation (ETD) Repository.
Blackmore, Sarah Elizabeth, "The effects of simulated parkinsonian rigidity on energy expenditure during gait" (2019). Electronic Theses and Dissertations. 2017.